HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS		QUICK SEARCH:   [advanced] Author: Keyword(s): Year:  Vol:  Page:

This Article Full Text (PDF) Alert me when this article is cited Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager Citing Articles Citing Articles via CrossRef Citing Articles via Google Scholar Google Scholar Articles by Závada, J. Search for Related Content PubMed PubMed Citation Articles by Závada, J. Agricola Articles by Závada, J.

A Host-cell DNA Function Involved in the Replication of Avian Tumour Viruses and of Fowl-plague Virus

Institute of Virology, Czechoslovak Academy of Science, Bratislava

The multiplication of fowl-plague virus was much less sensitive to actinomycin D in chick cells infected with avian myeloblastosis virus and in hamster cells, transformed by Rous sarcoma virus than in comparable normal cultures. In addition, reproduction of fowl-plague virus was less effectively suppressed by u.v.-irradiation of cells carrying avian myeloblastosis or Rous sarcoma virus than of control cultures. These findings could indicate that avian myeloblastosis and Rous sarcoma viruses induce a function of host-cell DNA, whose products can be utilized in reproduction of fowl-plague virus.

^* Present address: Imperial Cancer Research Fund, Lincoln's Inn Fields, London, W.C. 2

Received 18 September 1968; accepted 2 December 1968.