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HSV-1 Infection Inhibits Procollagen and Protein Secretion from Normal and Ataxia Telangiectasia Cultured Skin Fibroblasts

Department of Molecular Virology The Hebrew University-Hadassah Medical School, Jerusalem, Israel

Human skin fibroblasts derived from a healthy individual and from a child with the genetic disorder ataxia telangiectasia were infected with herpes simplex virus type 1 (HSV-1). The virus infection did not affect the synthesis of procollagen but inhibited its release from the cells.

^* Present address: Department of Biochemistry, University of Glasgow, Glasgow, Scotland.

Received 2 April 1980; accepted 5 June 1980.