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Second-site mutations in Borna disease virus overexpressing viral accessory protein X, by Marion Poenisch, Sandra Wille, Urs Schneider and Peter Staeheli

Journal of General Virology vol. 90, part 8, pp. 1932–1936

Supplementary Fig. S1. X protein of BDV-X5′ #2 with L12S and V13A substitutions lost the ability to interact with the viral P protein.

Supplementary Fig. S2. XK82E retains ability to block viral polymerase activity.

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