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Corrigendum for Law et al., J Gen Virol 83 (1) 209-222.
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Journal of General Virology (2002), 83, 1251.
© 2002 Society for General Microbiology

Corrigendum

Antibody-sensitive and antibody-resistant cell-to-cell spread by vaccinia virus: role of the A33R protein in antibody-resistant spread

Mansun Law, Ruth Hollinshead and Geoffrey L. Smith

Journal of General Virology (2002), 83209–222

Fig. 2. The original legend was found to be incorrect; the correct version is given below.

Fig. 2. (a) Cell-to-cell spread of VV in the presence of Ab. BS-C-1 cells were infected by WR or v{Delta}A56R at 20 p.f.u. per dish other mutants at 75 p.f.u. per well. Cells were overlaid with CMC in the absence of Ab (control) or in the presence of MAb 2D5 (diluted 1/500) Rb-WR2 or {alpha}-B5R antiserum (diluted 1/100) stained with crystal violet at 10 days p.i. (b–f) Quantification of plaque sizes in the presence or absence of Ab. (b) The diameters of plaques made by the different viruses in (a) were measured. The data represent the mean ±SEM of the diameter of six plaques selected randomly. An asterisk indicates that v{Delta}F13L plaques in the presence of Rb-WR2 were too faint to measure accurately. (c) The relative plaque size (diameter) of each mutant compared to WR is plotted. (d–f) The relative size of plaques formed by the mutants in the presence of MAb 2D5 (d) Rb-WR2 (e) or {alpha}-B5R (f) compared to control are shown.





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